Crește rata de supraviețuire | Cancer de ficat | Etapa 1 | Etapa 2 | Etapa 3 | Etapa 4

Crește rata de supraviețuire | Cancer de ficat | Etapa 1 | Etapa 2 | Etapa 3 | Etapa 4

Best adjuvant (assist) for chemotherapy | 1+1>487% |

Effectively improve chemotherapy effect, treatment, immunity |

Reduce side effects and recurrence | 

Overview / Relation / Abstract / Role / Principle / Action / Mechanism / Function / Work | 

Abstract / Summary / Overview of Apoptosis

Why do cells undergo apoptosis?

The relationship between cancer cells and apoptosis

Where are the weaknesses and symptoms of cancer cells?

Are cancer cells aggressive?

Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work)

Solamargine's major function mechanism:

Solamargine vs cancer

Best Chemotherapy Adjuvant  (1+1>478%) Effectively improve chemotherapy effect and cure



Best adjuvant (assist) for chemotherapy | 1+1>487%

Effectively improve chemotherapy effect, treatment, immunity 

Reduce side effects and recurrence 

Overview / Relation / Abstract / Role / Principle  / Action / Mechanism / Function / Work

Abstract / Summary / Overview of Apoptosis

Apoptosis.jpg

Overview of apoptosis

•Programmed cell death

•Apoptosis is a form of programmed cell death, or “cellular suicide.”

•Apoptosis is different from necrosis, in which cells die due to injury.

•Apoptosis removes cells during development, eliminates potentially cancerous and virus-infected cells, and maintains balance in the body.


Why do cells undergo apoptosis?

  • Basically, apoptosis is a general and convenient way to remove cells that should no longer be part of the organism.
  • Some cells are abnormal and could hurt the rest of the organism if they survive, such as cells with viral infections or DNA damage.
  • Apoptosis is part of development
  • In many organisms, programmed cell death is a normal part of development.


The relationship between cancer cells and apoptosis

Apoptosis can eliminate infected or cancerous cells.

When a cell’s DNA is damaged, it will typically detect the damage and try to repair it. 

If the damage is beyond repair, the cell will normally send itself into apoptosis, ensuring that it will not pass on its damaged DNA. 

When cells have DNA damage but fail to undergo apoptosis, they may be on the road to cancer.

However, “successful” cancer cells successfully evade the process of apoptosis.

This allows them to divide out of control and accumulate mutations (changes in their DNA).

Apoptosis is key to immune function

Apoptosis also plays an essential role in the development and maintenance of a healthy immune system. 


Where are the weaknesses and symptoms of cancer cells?

The symptoms of cancer cells are in the nucleus.

The nucleus controls the outer cytoplasm, cell composition, cell viability, etc.

DNA mutations also mutate in the nucleus.

Therefore, to treat cancer cells, we must first enter the nucleus.

Let the “regulatory cell gene” mechanism enter the nucleus to regulate


Are cancer cells aggressive?

After the action of Solamargine, the aggressiveness of cancer cells is alleviated.

So after using Solamargine, many patients feel that I am half better.

Although the tumor does not disappear quickly, patients feel that the degree of aggressiveness is reduced.


Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work)


sr-t100_apoptosis_mechanism005.jpg


Solamargine's major function mechanism:

When Solamargine enter,

Solamargine activates receptors that are turned off by cancer cells, allowing cancer cells to modulate again.

Solamargine modulates the anti-modulates genes of cancer cells, making cancer cells less resistant.

Reduced drug resistance

When cancer cells are less resistant to drugs, chemotherapy becomes more effective.

Solamargine modulates the mutated genes in cancer cells and then initiates cancer cell apoptosis to achieve anti-cancer effects.

Solamargine combined with which chemotherapy drugs are more effective in treating cancer cells?

Chemotherapy_01.jpg


Solamargine vs cancer

cell apoptosis.jpg

Solamargine vs cancer

The picture shows the death of cancer cells.

The black and black parts are cancer cell nuclei.

Even if the nucleus ruptures, the cancer cells will die.

The figure shows that cancer cells can cause death.


cancer cell apoptosis_01_800.jpg

The figure shows that cancer cells can cause death.

The figure shows that the death of lung cancer cells is relatively slow, and it will not be obvious until eight hours later.

The figure shows that the death of liver cancer cells is very obvious, even more obvious in eight hours.

The graph shows that breast cancer cells die faster. It was obvious from the beginning that breast cancer is easy to treat, and patients with breast cancer need not worry.



Best Chemotherapy Adjuvant (1+1>487%) Effectively improve chemotherapy effect and treatmentsolamargine vs cancer_lung cancer cell.jpg

ANTI-CANCER 

Patent protection in 32 nations. 

A comparison study showing Solamargine vs. other therapeutic drugs with respect to lung cancer cells.

solamargine vs cancer_breast cancer cell_01_800.jpg


A comparison study showing Solamargine vs. other chemotherapeutic drugs with respect to breast cancer cells

solamargine combined treatment therapy_01_800.jpg

SR-T100 combination therapy with effective result against breast cancer cells.


solamargine combined treatment therapy_03R12_800.jpg

Combination Therapy   |   Research results for lung cancer cells. 

A. Chemotherapy    (100μM), 16% of cancer cell apoptosis. 

B. Alone SM (4.8μM), 28% of cancer cell apoptosis. 

C. SM (4.80μM) + Chemotherapy (40μM), 66% of cancer cells apoptosis.  

D. SM (4.80μM) + Chemotherapy (100μM), 78% of cancer cell apoptosis.  

SM has a clearing effect better than Chemotherapy. 

The combined treatment of Solamargine and Chemotherapy significantly increased the apoptosis of lung cancer cells.  

SM (4.8μM) + Chemotherapy (40μM), increased from 16% to 66% (up to 4.125 times).  

SM (4.8μM) + Chemotherapy (100μM), increased from 16% to 78% (up to 4.875 times)  

Reorganized from: BBRC. Action of Solamargine on TNFs and drug-resistant human lung cancer cells 2004


justnow_02.jpg

The best solution for cancer cells

Solamargine Q&A (English)



Cancer Medical Care | Side Effects of Chemotherapy | Improve Anemia | Improve Low Red Blood Cells | Improve Low Hemoglobin (Decrease/Decrease/Decrease/Insufficiency) 

Cancer Medical Care | Chemotherapy by-products | Leukopenia | Neutropenia 

Cancer Medical Care | Chemotherapy Side Effects| Improve Bleeding| Improve Platelet Decrease| Improve Platelet Deficiency| Improve Platelet Low| Improve Thrombocytopenia 

Cancer Medical Care | Six indicators of physical health self-assessment: sleep, appetite, excretion, physical strength, mental strength, and psychology (emotion, mind) 
 


Chemotherapy | Increase cure rate 
Reduce cancer recurrence 
Apoptotic (Correct/ Guide) bad behavior 
Cure all diseases | Panacea | Package health 
How to Longevity? 
Want to Change? 
Life Above All | Release life is best for oneself (me / yourself) 
Eliminate misfortune! 
Good affinities with others. 
 
Beauty in Nature   



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Cancer de ficat | Rata de supraviețuire | Contents
Cancer de ficat | Rata de supraviețuire | Etymology and definitions
Cancer de ficat | Rata de supraviețuire | Signs and symptoms
Cancer de ficat | Rata de supraviețuire | Local symptoms
Cancer de ficat | Rata de supraviețuire | Systemic symptoms
Cancer de ficat | Rata de supraviețuire | Metastasis
Cancer de ficat | Rata de supraviețuire | Causes
Cancer de ficat | Rata de supraviețuire | Chemicals
Cancer de ficat | Rata de supraviețuire | Diet and exercise
Cancer de ficat | Rata de supraviețuire | Infection
Cancer de ficat | Rata de supraviețuire | Radiation
Cancer de ficat | Rata de supraviețuire | Heredity
Cancer de ficat | Rata de supraviețuire | Physical agents
Cancer de ficat | Rata de supraviețuire | Hormones
Cancer de ficat | Rata de supraviețuire | Autoimmune diseases
Cancer de ficat | Rata de supraviețuire | Pathophysiology
Cancer de ficat | Rata de supraviețuire | Genetics
Cancer de ficat | Rata de supraviețuire | Epigenetics
Cancer de ficat | Rata de supraviețuire | Metastasis
Cancer de ficat | Rata de supraviețuire | Metabolism
Cancer de ficat | Rata de supraviețuire | Diagnosis
Cancer de ficat | Rata de supraviețuire | Classification
Cancer de ficat | Rata de supraviețuire | Prevention
Cancer de ficat | Rata de supraviețuire | Dietary
Cancer de ficat | Rata de supraviețuire | Medication
Cancer de ficat | Rata de supraviețuire | Vaccination
Cancer de ficat | Rata de supraviețuire | Screening
Cancer de ficat | Rata de supraviețuire | Recommendations
Cancer de ficat | Rata de supraviețuire | Genetic testing
Cancer de ficat | Rata de supraviețuire | Management
Cancer de ficat | Rata de supraviețuire | Chemotherapy
Cancer de ficat | Rata de supraviețuire | Radiation
Cancer de ficat | Rata de supraviețuire | Surgery
Cancer de ficat | Rata de supraviețuire | Palliative care
Cancer de ficat | Rata de supraviețuire | Immunotherapy
Cancer de ficat | Rata de supraviețuire | Laser therapy
Cancer de ficat | Rata de supraviețuire | Alternative medicine
Cancer de ficat | Rata de supraviețuire | Prognosis
Cancer de ficat | Rata de supraviețuire | Epidemiology
Cancer de ficat | Rata de supraviețuire | History
Cancer de ficat | Rata de supraviețuire | Society and culture
Cancer de ficat | Rata de supraviețuire | Economic effect
Cancer de ficat | Rata de supraviețuire | Workplace
Cancer de ficat | Rata de supraviețuire | Research
Cancer de ficat | Rata de supraviețuire | Pregnancy
Cancer de ficat | Rata de supraviețuire | Other animals
Cancer de ficat | Rata de supraviețuire | Notes
Cancer de ficat | Rata de supraviețuire | Further reading
Cancer de ficat | Rata de supraviețuire | External links